Medicine

T. Ishikawa, K. Sonehara, K. Sonoda, Kenshi Hayashi, Koichi Kato, Satoshi Nagase, K. Kusano, T. Aiba, Minoru Horie, S. Ohno, Yukinori Okada, N. Makita

2026.1.26Journal of Arrhythmia

DOI: 10.1002/joa3.70273

Abstract

Rare pathogenic variations of desmosomal genes, particularly in plakophilin‐2 (PKP2) and desmoglein‐2 (DSG2), have been implicated in arrhythmogenic cardiomyopathy (ACM); however, their potential polygenic contribution remains unclear.

Citation format

ISHIKAWA, T., et al. A genome‐wide association study reveals desmoglein‐2 predominance in japanese arrhythmogenic cardiomyopathy. Journal of Arrhythmia, 2026, 42(1): e70273.