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Uric acid as a mediator of endothelial dysfunction, inflammation, and vascular disease.

J. Kanellis, D. Kang

2005SEMINARS IN NEPHROLOGY

DOI: 10.1016/j.semnephrol.2004.09.007

tlooto Summary

In vitro and in vivo findings suggest that uric acid may contribute to endothelial dysfunction by inducing antiproliferative effects on endothelium and impairing nitric oxide production.

Abstract

Abstract is not available.

Citation format

KANELLIS, J.; KANG, D. Uric acid as a mediator of endothelial dysfunction, inflammation, and vascular disease. SEMINARS IN NEPHROLOGY, 2005, 25 1: 39–42.